Weight LossBrainEnergyAnti-AgingLongevitySubcutaneous

MOTS-c

10mg Vial

Typical Dose

5–10 mg

Frequency

2–3x/week

Half-Life

Estimated 4–8 hours

Research-use dosing; specific study population not established.

MOTS-c Overview

Mechanism of Action

MOTS-c (Mitochondrial Open Reading Frame of the Twelve S rRNA type-c) is a 16-amino-acid peptide encoded in mitochondrial DNA — making it one of the few known mitochondria-derived signaling peptides. It activates AMPK (the master metabolic switch), enhances insulin sensitivity, and promotes glucose uptake independent of insulin. Remarkably, it is exercise-induced — its levels rise during physical activity.

Half-Life

Estimated 4–8 hours

Timing

Morning or pre-exercise

Injection Site

Subcutaneous, abdomen

Food

Can be taken fasted or with food

Common Stacks

  • NAD+ — Mitochondrial function synergy
  • AOD-9604 — Complementary fat metabolism

MOTS-c Dosing Protocol

Standard Protocol

8–12 weeks

10–15 mg, 2–3x/week

Often dosed on non-training days. Some protocols time it pre-exercise.

MOTS-c Expected Timeline

Week 1–2

Improved exercise endurance and recovery

Week 3–4

Enhanced metabolic flexibility; better blood sugar regulation

Week 6–8

Noticeable body composition improvement; reduced visceral fat

Week 8–12

Sustained metabolic optimization; improved mitochondrial function

MOTS-c Side Effects

Mild GI discomfort (uncommon)

Temporary muscle soreness at higher doses

Very well tolerated overall

MOTS-c Research Studies

The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance

Lee C, Zeng J, Drew BG, et al. — Cell Metab (2015)

Discovery paper published in Cell Metabolism demonstrating MOTS-c activates AMPK, prevents age-dependent and high-fat-diet-induced insulin resistance, and improves metabolic homeostasis in mice.

PubMed: 25738459

MOTS-c is an exercise-induced mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis

Reynolds JC, Lai RW, Woodhead JST, et al. — Nat Commun (2021)

Published in Nature Communications, showed MOTS-c levels increase with exercise and that MOTS-c treatment improved physical capacity in aging mice, acting as an exercise mimetic.

PubMed: 33397966

MOTS-c FAQ

Is MOTS-c an exercise replacement?

MOTS-c activates many of the same metabolic pathways as exercise (particularly AMPK activation and glucose uptake), earning it the label “exercise mimetic.” However, it’s best used as an adjunct to exercise, not a replacement. It can be especially valuable for those with limited exercise capacity.

How does MOTS-c differ from other metabolic peptides?

MOTS-c is unique because it’s encoded in mitochondrial DNA (not nuclear DNA like most peptides) and is the first identified mitochondrial-derived peptide with metabolic signaling function. It targets metabolic pathways at their source — the mitochondria.

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